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Effect of Collateral Puncture-bloodletting at JING Point on Expression of HSP70 Protein of Cerebral Cortex in Rats with Middle Cerebral Artery Occlusion
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DOI   10.11656/j.issn.1672-1519.2005.06.18
Key Words   collateral puncture-bloodletting;acute ischemic cerebral injury;rats;HSP protein
Author NameAffiliation
WANG Xiu-yun Tianjin University of TCM, Tianjin 300193, China 
LI Ji-sheng 武警医学院军事预防医学研究所, 天津, 300162 
LIU Gong-wang Tianjin University of TCM, Tianjin 300193, China 
郭义 Tianjin University of TCM, Tianjin 300193, China 
张果忠 武警医学院军事预防医学研究所, 天津, 300162 
潘荣菁 Tianjin University of TCM, Tianjin 300193, China 
王强 Tianjin University of TCM, Tianjin 300193, China 
李桂兰 Tianjin University of TCM, Tianjin 300193, China 
韩煜 Tianjin University of TCM, Tianjin 300193, China 
Abstract
    [Objective] To observe the brain-protective effects of collateral puncture-bloodletting at JING point on the expression of HSP protein of brain in rats with acute local cerebral ischemia.[Methods] The cerebral injury with acute local ischemia was induced by occlusion of the middle cerebral artery with blood concretion in rats. The expression of HSP protein in ischemic region of brain tissue was determined with immunohistochemistry and the intervention of bloodletting at JING point was observed.[Result] Acute cerebral ishemic injury resulted in the expression of HSP protein in the ischemic region of brain tissue and this effect increased gradually with time. Bloodletting at JING point can enhance the expression of HSP protein in initial stage of ischemia and increase the resistance of brain against the exacerbation of the ischemic injury.[Conclusion] Bloodletting at JING point may improve the anti-stress ability of brain tissue in ischemic field and the protective function of the brain effectively.

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