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Study on dampness-heat syndrome of jin ju (non-ischemic gangrene of diabetic foot) based on glucose and lipid metabolism |
Hits 1622 Download times 796 Received:November 16, 2019 |
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DOI
10.11656/j.issn.1672-1519.2020.03.14 |
Key Words
jin ju (non-ischemic gangrene of diabetic foot);dampness heat and excessive toxin syndrome;glucose and lipid metabolism |
Author Name | Affiliation | E-mail | LI Liangliang | Department of Vascular Anomalies, Shanghai University of Traditional Chinese Medicine, Shanghai TCM-Integrated Hospital, Shanghai 200082, China | | WANG Weiling | Department of Vascular Anomalies, Shanghai University of Traditional Chinese Medicine, Shanghai TCM-Integrated Hospital, Shanghai 200082, China | | ZHAO Cheng | Department of Vascular Anomalies, Shanghai University of Traditional Chinese Medicine, Shanghai TCM-Integrated Hospital, Shanghai 200082, China Clinical Laboratory, Shanghai TCM-Integrated Institute of Vascular Anomalies, Shanghai 200082, China | Chengzhao_79@163.com |
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Abstract
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[Objective] Study on glucose and lipid metabolism indexes of dampness-heat syndrome of jin ju (non-ischemic gangrene) in diabetic foot by modern testing techniques.[Methods] A total of 136 patients with jin ju (diabetic foot non-ischemic gangrene) were collected and compared for different syndrome types.[Results] The levels of FBG,HbA1c,2 hPBG,AGEs,ALB,TC,HDL-C in patients with dampness-heat syndrome were different from those in other syndromes of jin ju (non-ischemic gangrene) (P<0.05). And the levels of CRP and sVCAM-1 in patients with dampness-heat syndrome were significantly different from those in other syndromes of jin ju (non-ischemic gangrene) (P<0.01).[Conclusion] The cause of the formation of jin ju (non-ischemic gangrene) dampness-heat toxin in diabetic foot is related to hyperglycemia and persistent inflammatory response caused by disorders of glucose and lipid metabolism in low-nutrition conditions. |
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